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Although we were the first group to describe the phenomenon in the veterinary literature (Lisciandro 2016b), all the credit goes to Dr Scott Johnson, of Austin, Texas. When Dr Johnson took our Global FAST course in 2010, he remarked that he had observed hemoabdomen in anaphylactic dogs, and that we should start looking during AFAST. We heeded his suggestion and have seen close to 100 canine anaphylactic dogs with positive fluid scores and dozens with confirmed hemoabdomen that responded to medical treatment (Lisciandro 2014a, 2016b; Hnatusko et al. 2019). Importantly, these are witnessed or unwitnessed events likely caused by a presumed single Hymenoptera species envenomation (not massive bee envenomation). The great majority of anaphylactic dogs have no obvious cutaneous signs (Lisciandro 2016b; Hnatusko et al. 2019).

Heparin, a clinically tangible constituent of mast cells, likely plays a major role, thus the addition of “heparin‐induced” by the author to its descriptor (Lisciandro 2016b). The importance of recognizing this AX‐related heparin‐induced hemoabdomen is that these dogs are medically treated (Lisciandro 2014a, 2016b; Caldwell et al. 2018; Birkbeck et al. 2019; Hnatusko et al. 2019) and inadvertently taking these dogs to surgery will anecdotally result in a fatal outcome (Lisciandro 2014a, 2016b; Hnatusko et al. 2019). Interestingly, in a published case series of 432 dogs with hemoabdomen, 86 were operated, but only 83 were included in the study because the three that were excluded had no histopathological diagnosis (Lux et al. 2013). One has to wonder if these three dogs were AX‐related heparin‐induced hemoabdomen cases.