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Amar S, Chung KM. Influence of hormonal variation on the periodontium in women. Periodontol 2000 1994; 6(6):79–87

Berkovitz BK. Periodontal ligament: structural and clinical correlates. Dent Update 2004; 31(1):46–50, 52, 54

Lovegrove JM. Dental plaque revisited: bacteria associated with periodontal disease. J N Z Soc Periodontol 2004; 87(87):7–21

Lyngstadaas SP, Wohlfahrt JC, Brookes SJ, Paine ML, Snead ML, Reseland JE. Enamel matrix proteins; old molecules for new applications. Orthod Craniofac Res 2009; 12(3):243–253

Marsh PD, Martin MV, Lewis MAO, Williams DW. Oral microbiology. London: Churchill Livingstone; 2009

Mineoka T, Awano S, Rikimaru T, et al. Site-specific development of periodontal disease is associated with increased levels of Porphyromonas gingivalis, Treponema denticola, and Tannerella forsythia in subgingival plaque. J Periodontol 2008; 79(4):670–676

Scott DA, Krauss J. Neutrophils in periodontal inflammation. Front Oral Biol. 2012; 15:56–83

Wesselink PR, Beertsen W. The prevalence and distribution of rests of Malassez in the mouse molar and their possible role in repair and maintenance of the periodontal ligament. Arch Oral Biol 1993; 38(5):399–403

Xiong J, Mrozik K, Gronthos S, Bartold PM. Epithelial cell rests of Malassez contain unique stem cell populations capable of undergoing epithelial-mesenchymal transition. Stem Cells Dev 2012; 21(11):2012–2025

Applied Oral Physiology

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