Читать книгу Canine and Feline Epilepsy - Luisa De Risio - Страница 109

The suspicion of toxin exposure is often based on the history and the onset of acute neurological signs (including excitation and hyperactivity or obtundation, stupor, coma, muscle tremors and fasciculations, seizures and ataxia) often associated with vomiting, diarrhoea, salivation, bronchoconstriction, bradycardia or tachycardia and hyperthermia. The source of intoxication is not always obvious to the pet owner, and therefore veterinarians should be proactive in asking questions and mentioning possible sources of intoxication any time the clinical presentation raises the suspicion of toxin exposure. Seizure can occur in clusters or as status epilepticus (e.g. organophosphates, strychnine, mycotoxins) or less commonly may be isolated and recurrent (e.g. lead) (O’Brien, 1998).