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De Quervain’s disease is a specific wrist tendinopathy involving the entrapment of the tendons of the extensor pollicis brevis and abductor pollicis longus. Figure 2.4 illustrates the region associated with the development of de Quervain’s syndrome. Patients with de Quervain’s disease (as well as trigger finger) tend to exhibit a lack of inflammation in the affected region (Clarke, 1998; Sbernardori & Bandiera, 2007). Instead, there appears to be a breakdown of tissues of the gliding layer of the tendon sheath. Examination of tendons tends to reveal nodularity and tendon fraying, which is thought to be secondary to impingement by the damaged sheath. Both disorders often exhibit the development of fibrotic tissue. In the case of de Quervain’s syndrome, deposition of fibrotic tissue in the extensor retinaculum can be thickened up to five times greater than the thickness in control tissues (Clarke, 1998). Overall, signs point to a process of tissue degeneration as opposed to an inflammatory response, with the degeneration resulting from abnormal mechanical stress being imposed on the tendon and the tendon sheath (McCauliffe, 2010).