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24. CELLULAR INJURY

Оглавление

Keyboard Associations

•Aplasia ⇔ Agenesis

•Anaplasia ⇔ Lack of consistent differentiation

•Hypoplasia ⇔ slow genesis

•Metaplasia ⇔ replacement of tissues

•Hypertrophy ⇔ Size

•Hyperplasia ⇔ Number

•Irreversible damage indicators ⇔ AST, CPK, LDH

•Autolysis ⇔ Degradation by internal enzymes

•Heterolysis ⇔ Degradation by extrinsic enzymes

•Pyknosis ⇔ Nuclear shrinkage

•Karyorrhexis ⇔ Chromatin fragmentation

•Karyolysis ⇔ Dissolution of pyknotic nucleus

•Apoptosis ⇔ Councilman body of viral hepatitis

•Coagulation necrosis ⇔ nuclear changes

•Liquefaction necrosis ⇔ Liquefied necrotic tissues (Brain)

•Caseous necrosis ⇔ Soft, cheesy (TB)

•Gangrenous Necrosis ⇔ Wet/dry

•Fibrinoid necrosis ⇔ Arterial fibrin-like deposition

•Traumatic Fat necrosis ⇔ Breast

•Enzymatic Fat necrosis ⇔ Pancreas

•Lipofuscin ⇔ Wear/Tear aging

•Hemosiderin ⇔ Red cell destruction

•Increased Melanin ⇔ sun tanning

•Decreased Melanin ⇔ Vitiligo

•Free radical ⇔ Lipid peroxidation

•Carbon ⇔ Anthracosis

•Lead poisoning (Plumbism) ⇔ RBC Basophilic stippling

•Methanol poisoning ⇔ CNS edema, blindness

•Carbon monoxide ⇔ Cherry red skin

Primary Care for COMLEX Level 1

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