Читать книгу Primary Care for COMLEX Level 1 - Dr. Jahan Eftekar - Страница 27
24. CELLULAR INJURY
ОглавлениеKeyboard Associations
•Aplasia ⇔ Agenesis
•Anaplasia ⇔ Lack of consistent differentiation
•Hypoplasia ⇔ slow genesis
•Metaplasia ⇔ replacement of tissues
•Hypertrophy ⇔ Size
•Hyperplasia ⇔ Number
•Irreversible damage indicators ⇔ AST, CPK, LDH
•Autolysis ⇔ Degradation by internal enzymes
•Heterolysis ⇔ Degradation by extrinsic enzymes
•Pyknosis ⇔ Nuclear shrinkage
•Karyorrhexis ⇔ Chromatin fragmentation
•Karyolysis ⇔ Dissolution of pyknotic nucleus
•Apoptosis ⇔ Councilman body of viral hepatitis
•Coagulation necrosis ⇔ nuclear changes
•Liquefaction necrosis ⇔ Liquefied necrotic tissues (Brain)
•Caseous necrosis ⇔ Soft, cheesy (TB)
•Gangrenous Necrosis ⇔ Wet/dry
•Fibrinoid necrosis ⇔ Arterial fibrin-like deposition
•Traumatic Fat necrosis ⇔ Breast
•Enzymatic Fat necrosis ⇔ Pancreas
•Lipofuscin ⇔ Wear/Tear aging
•Hemosiderin ⇔ Red cell destruction
•Increased Melanin ⇔ sun tanning
•Decreased Melanin ⇔ Vitiligo
•Free radical ⇔ Lipid peroxidation
•Carbon ⇔ Anthracosis
•Lead poisoning (Plumbism) ⇔ RBC Basophilic stippling
•Methanol poisoning ⇔ CNS edema, blindness
•Carbon monoxide ⇔ Cherry red skin