Читать книгу Large Animal Neurology - Joe Mayhew - Страница 97

Starvation and disorders associated with inadequate vitamin A, vitamin D, calcium, and phosphorus intake can be associated with vertebral fractures. Thiamine deficiency can cause cerebrocortical neuronal necrosis in ruminants, hemorrhagic necrosis of white matter in felidae and axonal degeneration in horses. Lesions of cell death tend to be symmetrical with nutritional diseases. Of course, dietary imbalances involving calcium, phosphorus, vitamin A, vitamin D, copper, etc., can result in neurocrania and vertebral defects that impinge on CNS and PNS structures asymmetrically, especially when secondary trauma plays a role.

Antioxidant failure effects various syndromes depending on age, and species especially. Thus, as exemplified by vitamin E deficiency, this may be cardiomyopathy in growing pigs, myopathy in young ruminants, neuroaxonal dystrophy in adolescent horses, or motor neuron disease in older horses.