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Interruption of the blood supply results in hemorrhagic or ischemic infarction and tissue necrosis (Figure 4.3), with neutrophilic and then monocytic phagocytosis of debris. Emboli and thrombi may be septic, parasitic, or fibrocartilaginous. More generalized infarction may occur with cardiopulmonary failure, disseminated clotting abnormalities, and septicemia. Hypertension, perhaps on a compartmental basis, may play a role in the vascular accidents associated with the neonatal hypoxic and ischemic encephalopathy seen most often in foals.³⁰